Intracerebroventricular gisenosides are antinociceptive in proinflammatory cytokine-induced pain behaviors of mice
YJ Seo, MS Kwon, HW Choi, JE Jang, JK Lee… - Archives of pharmacal …, 2008 - Springer
Several ginsenoside (R b1, R b2, R c, R d, R e, R f, R g1 and R g3) are neuroprotective and
antinociceptive agents. In this study, we assessed the effects of these ginsenosides
following intracerebroventricular (icv) administration on the nociceptive behaviors induced
by intrathecal injection of pro-inflammatory cytokines (tumor necrosis factor-a (TNF-α),
interleukin-1β (IL-1β), and interferon-γ (IFN-γ)). The ginsenosides, R b1, R b2, R c, R d, R e,
R f and R g1, significantly attenuated the nociceptive behavior induced by TNF-α, IL-1β, and …
antinociceptive agents. In this study, we assessed the effects of these ginsenosides
following intracerebroventricular (icv) administration on the nociceptive behaviors induced
by intrathecal injection of pro-inflammatory cytokines (tumor necrosis factor-a (TNF-α),
interleukin-1β (IL-1β), and interferon-γ (IFN-γ)). The ginsenosides, R b1, R b2, R c, R d, R e,
R f and R g1, significantly attenuated the nociceptive behavior induced by TNF-α, IL-1β, and …
Intracerebroventricular gisenosides are antinociceptive in proinflammatory cytokine-induced pain behaviors of mice.
SYJ Seo YoungJun, KMS Kwon MinSoo… - 2008 - cabidigitallibrary.org
Abstract Several ginsenoside (Rb1, Rb2, Rc, Rd, Re, Rf, Rg1 and Rg3) are neuroprotective
and antinociceptive agents. In this study, we assessed the effects of these ginsenosides
following intracerebroventricular (icv) administration on the nociceptive behaviors induced
by intrathecal injection of pro-inflammatory cytokines (tumor necrosis factor-a (TNF-α),
interleukin-1β (IL-1β), and interferon-γ (IFN-γ)). The ginsenosides, Rb1, Rb2, Rc, Rd, Re, Rf
and Rg1, significantly attenuated the nociceptive behavior induced by TNF-α, IL-1β, and IFN …
and antinociceptive agents. In this study, we assessed the effects of these ginsenosides
following intracerebroventricular (icv) administration on the nociceptive behaviors induced
by intrathecal injection of pro-inflammatory cytokines (tumor necrosis factor-a (TNF-α),
interleukin-1β (IL-1β), and interferon-γ (IFN-γ)). The ginsenosides, Rb1, Rb2, Rc, Rd, Re, Rf
and Rg1, significantly attenuated the nociceptive behavior induced by TNF-α, IL-1β, and IFN …
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